KRAS insertion mutations are oncogenic and exhibit distinct functional properties.

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作者： Yasmine,White ^[1] ; Aditi,Bagchi ^[2] ; Jessica,Van Ziffle ^[3] ; Anagha,Inguva ^[4] ; Gideon,Bollag ^[1] ; Chao,Zhang ^[5] ; Heidi,Carias ^[5] ; David,Dickens ^[5] ; Mignon,Loh ^[2] ; Kevin,Shannon ^[1] ; Ari J,Firestone ^[6]

作者单位： Department of Pediatrics, University of California, 1450 3rd St, San Francisco, California 94158, USA. ^[1] Department of Pediatrics, Division of Pediatric Hematology/Oncology and Bone Marrow Transplantation; Helen DeVos Children's Hospital/Spectrum Health Medical Group, 100 Michigan St, Grand Rapids, Michigan 49503, USA. ^[2] Van Andel Institute Graduate School, 333 Bostwick Ave N.E., Grand Rapids, Michigan 49503, USA. ^[3] Department of Pathology, University of California, 2340 Sutter St, San Francisco, California 94115, USA. ^[4] Plexxikon Inc, 91 Bolivar Dr, Berkeley, California 94710, USA. ^[5] Helen Diller Family Comprehensive Cancer Center, University of California, 1450 3rd St, San Francisco, California 94158, USA. ^[6]

主题词动物(Animals);儿童, 学龄前(Child, Preschool);GTP酶激活蛋白质类(GTPase-Activating Proteins);肝细胞(Hepatocytes);人类(Humans);白血病, 粒-单核细胞, 幼年型(Leukemia, Myelomonocytic, Juvenile);小鼠(Mice);诱变, 插入(Mutagenesis, Insertional);突变(Mutation);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);原癌基因蛋白质类p21(ras)(Proto-Oncogene Proteins p21(ras));串联重复序列(Tandem Repeat Sequences);肿瘤干细胞测定(Tumor Stem Cell Assay)

DOI 10.1038/ncomms10647

PMID 26854029

发布时间 2018-11-13

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Nature communications

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KRAS insertion mutations are oncogenic and exhibit distinct functional properties.

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KRAS insertion mutations are oncogenic and exhibit distinct functional properties.

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