换一批
换一批FoxO3 inactivation promotes human cholangiocarcinoma tumorigenesis and chemoresistance through Keap1-Nrf2 signaling.
第一作者：
Li,Guan
第一单位：
State Key Laboratory of Cellular Stress Biology, Innovation Center for Cell Signaling Network, School of Life Sciences, Xiamen University, Xiamen, Fujian 361102, China.
作者：
主题词
动物(Animals);胆管肿瘤(Bile Duct Neoplasms);细胞系, 肿瘤(Cell Line, Tumor);胆管上皮癌(Cholangiocarcinoma);减量调节(Down-Regulation);抗药性, 肿瘤(Drug Resistance, Neoplasm);女(雌)性(Female);基因表达调控(Gene Expression Regulation);人类(Humans);小鼠, 裸(Mice, Nude);NF-E2相关因子2(NF-E2-Related Factor 2);氧化性应激(Oxidative Stress);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);随机分配(Random Allocation);交叉感知受体(Receptor Cross-Talk);信号传导(Signal Transduction);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1002/hep.28496
PMID
26857210
发布时间
2018-01-18
- 浏览10
Hepatology (Baltimore, Md.)
1914-27页
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