The binding capability of plasma phospholipid transfer protein, but not HDL pool size, is critical to repress LPS induced inflammation.

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作者： Yang,Yu ^[1] ; Yingjie,Cui ^[2] ; Yanan,Zhao ^[1] ; Shuai,Liu ^[1] ; Guohua,Song ^[1] ; Peng,Jiao ^[1] ; Bin,Li ^[2] ; Tian,Luo ^[1] ; Shoudong,Guo ^[1] ; Xiangjian,Zhang ^[1] ; Hao,Wang ^[1] ; Xian-Cheng,Jiang ^[3] ; Shucun,Qin ^[1]

作者单位： Key Laboratory of Atherosclerosis in Universities of Shandong and Institute of Atherosclerosis, Taishan Medical University, Taian, China, 271000. ^[1] School of Basic Medicine, Taishan Medical University, Tai'an, China, 271000. ^[2] Hebei Collaborative Innovation Center for Cardio-cerebrovascular Disease and Hebei Key Laboratory of Vascular Homeostasis, Shijiazhuang, China, 050000. ^[3] Department of Anatomy and Cell Biology, SUNY Downstate Medical Center, New York, USA, 11203. ^[4]

主题词动物(Animals);骨髓细胞(Bone Marrow Cells);细胞系(Cell Line);细胞因子类(Cytokines);内毒素血症(Endotoxemia);炎症(Inflammation);脂多糖类(Lipopolysaccharides);脂蛋白类, HDL(Lipoproteins, HDL);巨噬细胞(Macrophages);男(雄)性(Male);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);磷脂转移蛋白质类(Phospholipid Transfer Proteins)

DOI 10.1038/srep20845

PMID 26857615

发布时间 2018-11-13

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The binding capability of plasma phospholipid transfer protein, but not HDL pool size, is critical to repress LPS induced inflammation.

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The binding capability of plasma phospholipid transfer protein, but not HDL pool size, is critical to repress LPS induced inflammation.

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