Mechanisms and consequences of ATMIN repression in hypoxic conditions: roles for p53 and HIF-1.

导出 LinkOut

翻译打印收藏纠错

作者： Katarzyna B,Leszczynska ^[1] ; Eva-Leonne,Göttgens ^[1] ; Deborah,Biasoli ^[1] ; Monica M,Olcina ^[2] ; Jonathan,Ient ^[1] ; Selvakumar,Anbalagan ^[1] ; Stephan,Bernhardt ^[1] ; Amato J,Giaccia ^[2] ; Ester M,Hammond ^[1]

作者单位： Cancer Research UK and Medical Research Council Oxford Institute for Radiation Oncology, Department of Oncology, The University of Oxford, Oxford, OX3 7DQ, UK. ^[1] Division of Cancer and Radiation Oncology, Department of Radiation Oncology, Stanford University, Stanford, California 94305, USA. ^[2]

主题词细胞低氧(Cell Hypoxia);细胞系(Cell Line);胞浆动力蛋白类(Cytoplasmic Dyneins);基因表达调控(Gene Expression Regulation);人类(Humans);缺氧诱导因子1, α亚基(Hypoxia-Inducible Factor 1, alpha Subunit);转录因子(Transcription Factors);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53)

DOI 10.1038/srep21698

PMID 26875667

发布时间 2024-05-24

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览4

Scientific reports

2016年6卷

21698页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Mechanisms and consequences of ATMIN repression in hypoxic conditions: roles for p53 and HIF-1.

Scientific reports

相似文献

Scientific reports

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Mechanisms and consequences of ATMIN repression in hypoxic conditions: roles for p53 and HIF-1.

Scientific reports

相似文献

Scientific reports

相关期刊

检索历史清除