Sustained Endocannabinoid Signaling Compromises Decidual Function and Promotes Inflammation-induced Preterm Birth.
第一作者:
Xiaofei,Sun
第一单位:
From the Division of Reproductive Sciences, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio 45229 and xiaofei.sun@cchmc.org.
作者:
主题词
酰胺基水解酶类(Amidohydrolases);动物(Animals);花生四烯酸类(Arachidonic Acids);细胞, 培养的(Cells, Cultured);蜕膜(Decidua);内源性大麻酚类(Endocannabinoids);女(雌)性(Female);基因缺失(Gene Deletion);炎症(Inflammation);脂多糖类(Lipopolysaccharides);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);小鼠(Mice);多不饱和脂肪酸酰胺类(Polyunsaturated Alkamides);早产(Premature Birth);信号传导(Signal Transduction)
DOI
10.1074/jbc.M115.707836
PMID
26900150
发布时间
2021-02-05
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