5'-Monophosphate-activated protein kinase (AMPK) improves autophagic activity in diabetes and diabetic complications.
作者:
关键词
ACC, carboxylaseADP, adenosine diphosphateAMP, adenosine monophosphateAMP-activated protein kinasesAMPK, 5′-monophosphate-activated protein kinaseATP, adenosine triphosphateAdipoR, adiponectin receptorsAutophagyCaMKK, Ca2+ calmodulin-dependent protein kinase kinaseDEPTOR, DEP domain-containing mTOR-interacting proteinDM, Diabetes mellitusDN, Diabetic nephropathyDiabetesDiabetic complicationsERK, extracellular signal-regulated kinaseFoxO, forkhead box class OGFRs, glomerular filtration ratesIKK, IκB kinaseJLDG, Jinlida granuleJNK, janus kinaseLC3, light chain 3LKB1, liver kinase B1PKC, protein kinase CPRAS40, proline-rich Akt substrate 40?kDaRAPTOR, regulator associated protein of mTORSOGA, suppressor of glucose form autophagySQSTM1, sequestosome 1STZ, streptozotocinTSC, tuberous sclerosis complexULK1, Unc-51-like kinase 1VPS34, vacuolar protein-sorting 34mTOR, mammalian target of rapamycinmTORC1, mammalian target of rapamycin (mTOR) complex 1
DOI
10.1016/j.apsb.2015.07.009
PMID
26904395
发布时间
2020-09-30
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Acta pharmaceutica Sinica. B
2016年6卷1期
20-5页
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