Molecular validation of the precision-cut kidney slice (PCKS) model of renal fibrosis through assessment of TGF-β1-induced Smad and p38/ERK signaling.
第一作者:
Shouhua,Zhang
第一单位:
Department of General Surgery, The Second Affiliated Hospital of Nanchang University, Nanchang, Jiangxi Province, China; Department of General Surgery, Jiangxi Children's Hospital, Nanchang, Jiangxi Province, China.
作者:
主题词
主动转运, 细胞核(Active Transport, Cell Nucleus);动物(Animals);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);纤维化(Fibrosis);人类(Humans);肾(Kidney);肾疾病(Kidney Diseases);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);器官培养技术(Organ Culture Techniques);信号传导(Signal Transduction);Smad2蛋白质(Smad2 Protein);Smad3蛋白质(Smad3 Protein);转化生长因子β1(Transforming Growth Factor beta1);增量调节(Up-Regulation);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.intimp.2016.01.026
PMID
26919280
发布时间
2019-12-10
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