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Creatine salts provide neuroprotection even after partial impairment of the creatine transporter.

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第一作者： E,Adriano

第一单位： Department of Neuroscience, Ophthalmology, Genetics, Maternal-Infantile Sciences, University of Genova, Largo Paolo Daneo 3, 16132 Genova, Italy. Electronic address: adriano@neurologia.unige.it.

作者： E,Adriano ^[1] ; P,Garbati ^[2] ; A,Salis ^[3] ; G,Damonte ^[4] ; E,Millo ^[4] ; M,Balestrino ^[2]

作者单位： Department of Neuroscience, Ophthalmology, Genetics, Maternal-Infantile Sciences, University of Genova, Largo Paolo Daneo 3, 16132 Genova, Italy. Electronic address: adriano@neurologia.unige.it. ^[1] Department of Neuroscience, Ophthalmology, Genetics, Maternal-Infantile Sciences, University of Genova, Largo Paolo Daneo 3, 16132 Genova, Italy. ^[2] Department of Hearth Environmental and Life Science (DISTAV), University of Genova, Corso Europa 26, 16132 Genova, Italy; Center of Excellence for Biomedical Research, University of Genova, Viale Benedetto XV 5, 16132 Genova, Italy. ^[3] Department of Experimental Medicine, Section of Biochemistry, University of Genova, Viale Benedetto XV 1, 16132 Genova, Italy; Center of Excellence for Biomedical Research, University of Genova, Viale Benedetto XV 5, 16132 Genova, Italy. ^[4]

关键词 creatine creatine ascorbate creatine gluconate creatine glucose creatine transporter deficiency neuroprotection