换一批Prolyl hydroxylase domain 2 deficiency promotes skeletal muscle fiber-type transition via a calcineurin/NFATc1-dependent pathway.
第一作者：
Junchul,Shin
第一单位：
Department of Medicine & Science in Sport & Exercise, Tohoku University School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi 980-8575 Japan.
作者：
医学主题词
动物(Animals);钙神经素(Calcineurin);毛细血管(Capillaries);细胞低氧(Cell Hypoxia);细胞系(Cell Line);基因型(Genotype);羟基化(Hydroxylation);缺氧诱导因子1, α亚基(Hypoxia-Inducible Factor 1, alpha Subunit);小鼠, 基因敲除(Mice, Knockout);肌纤维, 慢缩(Muscle Fibers, Slow-Twitch);肌, 骨骼(Muscle, Skeletal);NFATC转录因子类(NFATC Transcription Factors);新生血管化, 生理性(Neovascularization, Physiologic);表型(Phenotype);蛋白质稳定性(Protein Stability);信号传导(Signal Transduction);他罗利姆(Tacrolimus);时间因素(Time Factors);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1186/s13395-016-0079-5
PMID
26949511
发布时间
2018-11-13
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