换一批
换一批Synergy of TLR3 and 7 ligands significantly enhances function of DCs to present inactivated PRRSV antigen through TRIF/MyD88-NF-κB signaling pathway.
第一作者：
Yue,Hu
第一单位：
Key Laboratory of animal biotechnology and disease control and prevention of Shandong Province, College of Animal Science and Veterinary Medicine, Shandong Agricultural University, Tai'an 271018, China.
作者：
主题词
衔接蛋白质类, 膜泡运输(Adaptor Proteins, Vesicular Transport);动物(Animals);抗原, 病毒(Antigens, Viral);CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);CD8阳性T淋巴细胞(CD8-Positive T-Lymphocytes);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);细胞因子类(Cytokines);树突细胞(Dendritic Cells);女(雌)性(Female);干扰素γ(Interferon-gamma);白细胞介素4(Interleukin-4);配体(Ligands);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);髓样分化因子88(Myeloid Differentiation Factor 88);NF-κB(NF-kappa B);猪呼吸与生殖综合征病毒(Porcine respiratory and reproductive syndrome virus);信号传导(Signal Transduction);猪(Swine);T淋巴细胞(T-Lymphocytes);Toll样受体3(Toll-Like Receptor 3);Toll样受体7(Toll-Like Receptor 7)
DOI
10.1038/srep23977
PMID
27046485
发布时间
2018-11-13
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