Analysis of single nucleotide polymorphisms implicate mTOR signalling in the development of new-onset diabetes after transplantation.

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作者： S,Chand ^[1] ; A J,McKnight ^[2] ; S,Shabir ^[1] ; W,Chan ^[3] ; J A,McCaughan ^[2] ; A P,Maxwell ^[2] ; L,Harper ^[1] ; R,Borrows ^[1]

作者单位： Department of Nephrology and Kidney Transplantation, Queen Elizabeth Hospital, Birmingham B15 2WB, United Kingdom; Centre for Translational Inflammation Research, University of Birmingham, Birmingham B15 2WB, United Kingdom. ^[1] Regional Nephrology Unit, Belfast City Hospital, Belfast BT9 7AB, Northern Ireland. ^[2] Department of Nephrology and Kidney Transplantation, Queen Elizabeth Hospital, Birmingham B15 2WB, United Kingdom. ^[3]

关键词 ATF6, Activated transcription factor BMI, Body mass index GWAS, Genome-wide association study HLA, Human leucocyte antigen HNF4, Hepatocyte nuclear factor 4 NODAT, New-onset diabetes after transplantation New-onset diabetes after transplantation PI3, Phospho-inositide 3-kinase PPARGC1α, Peroxisome proliferator-activated receptor gamma co-activator 1 alpha PPARy, Peroxisome proliferator-activated receptor gamma PRDM14, PR domain zinc protein 14 SNP, Single nucleotide polymorphism mTOR mTOR, Mammalian target of rapamycin single nucleotide polymorphisms

DOI 10.1016/j.bbacli.2015.12.004

PMID 27051588

发布时间 2022-03-10

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Analysis of single nucleotide polymorphisms implicate mTOR signalling in the development of new-onset diabetes after transplantation.

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Analysis of single nucleotide polymorphisms implicate mTOR signalling in the development of new-onset diabetes after transplantation.

BBA clinical

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BBA clinical

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