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Pharmacological reversal of a pain phenotype in iPSC-derived sensory neurons and patients with inherited erythromelalgia.

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第一作者： Lishuang,Cao

第一单位： Pfizer Neuroscience and Pain Research Unit, The Portway Building, Granta Park, Cambridge CB21 6GS, UK.

作者： Lishuang,Cao ^[1] ; Aoibhinn,McDonnell ^[1] ; Anja,Nitzsche ^[1] ; Aristos,Alexandrou ^[1] ; Pierre-Philippe,Saintot ^[1] ; Alexandre J C,Loucif ^[1] ; Adam R,Brown ^[1] ; Gareth,Young ^[1] ; Malgorzata,Mis ^[2] ; Andrew,Randall ^[3] ; Stephen G,Waxman ^[4] ; Philip,Stanley ^[1] ; Simon,Kirby ^[1] ; Sanela,Tarabar ^[5] ; Alex,Gutteridge ^[1] ; Richard,Butt ^[1] ; Ruth M,McKernan ^[1] ; Paul,Whiting ^[1] ; Zahid,Ali ^[1] ; James,Bilsland ^[6] ; Edward B,Stevens ^[6]

作者单位： Pfizer Neuroscience and Pain Research Unit, The Portway Building, Granta Park, Cambridge CB21 6GS, UK. ^[1] University of Bristol, School of Physiology, Pharmacology, and Neuroscience, Bristol BS8 1TD, UK. ^[2] Hatherly College of Life and Environmental Sciences, University of Exeter, Prince of Wales Road, Exeter EX4 4PS, UK. ^[3] Yale Center for Neuroscience and Regeneration Research, Veterans Affairs Medical Center, 950 Campbell Avenue, Building 34,West Haven, CT 06516, USA. ^[4] Pfizer, 1 Howe Street, New Haven, CT 06511, USA. ^[5] Pfizer Neuroscience and Pain Research Unit, The Portway Building, Granta Park, Cambridge CB21 6GS, UK. j.bilsland@ucl.ac.uk edward.stevens@pfizer.com. ^[6]