换一批Reciprocal activation between STAT3 and miR-181b regulates the proliferation of esophageal cancer stem-like cells via the CYLD pathway.
第一作者：
Dan-Dan,Xu
第一单位：
College of Life Science and Technology, Jinan University, 510632, Guangzhou, P.R. China.;Key laboratory of Bioengineering medicine of Guangdong Province, Jinan University, 510632, Guangzhou, P.R. China.
作者：
主题词
3'非翻译区(3' Untranslated Regions);动物(Animals);细胞凋亡(Apoptosis);结合部位(Binding Sites);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);食管肿瘤(Esophageal Neoplasms);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);小鼠(Mice);微RNAs(MicroRNAs);肿瘤干细胞(Neoplastic Stem Cells);RNA干扰(RNA Interference);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction);球形体, 细胞(Spheroids, Cellular);肿瘤细胞, 培养的(Tumor Cells, Cultured);肿瘤干细胞测定(Tumor Stem Cell Assay);肿瘤抑制蛋白质类(Tumor Suppressor Proteins);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays)
DOI
10.1186/s12943-016-0521-7
PMID
27189061
发布时间
2018-11-13
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