Recurrent, truncating SOX9 mutations are associated with SOX9 overexpression, KRAS mutation, and TP53 wild type status in colorectal carcinoma.
第一作者:
Breanna M,Javier
第一单位:
Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, NY, USA.;Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
作者:
医学主题词
成年人(Adult);老年人(Aged);结直肠肿瘤(Colorectal Neoplasms);女(雌)性(Female);人类(Humans);Kaplan-Meiers评估(Kaplan-Meier Estimate);男(雄)性(Male);中年人(Middle Aged);突变(Mutation);原癌基因蛋白质类p21(ras)(Proto-Oncogene Proteins p21(ras));SOX9转录因子类(SOX9 Transcription Factor);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53)
DOI
10.18632/oncotarget.9682
PMID
27248473
发布时间
2018-11-13
- 浏览9
Oncotarget
50875-50882页
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