A metabolic intermediate of the fructose-asparagine utilization pathway inhibits growth of a Salmonella fraB mutant.

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作者： Anice,Sabag-Daigle ^[1] ; Henry M,Blunk ^[2] ; Anindita,Sengupta ^[3] ; Jikang,Wu ^[4] ; Alexander J,Bogard ^[4] ; Mohamed M,Ali ^[4] ; Christopher,Stahl ^[2] ; Vicki H,Wysocki ^[3] ; Venkat,Gopalan ^[5] ; Edward J,Behrman ^[3] ; Brian M M,Ahmer ^[4]

作者单位： Department of Microbial Infection and Immunity, The Ohio State University, Columbus, OH 43210, USA. ^[1] Center for Microbial Interface Biology, The Ohio State University, Columbus, OH 43210, USA. ^[2] Department of Microbiology, The Ohio State University, Columbus, OH 43210, USA. ^[3] Department of Chemistry and Biochemistry, The Ohio State University, Columbus, OH 43210, USA. ^[4] Department of Medical Microbiology and Immunology, Faculty of Medicine, Mansoura University, Mansoura, Egypt. ^[5]

主题词动物(Animals);天冬酰胺(Asparagine);天冬氨酸(Aspartic Acid);细菌蛋白质类(Bacterial Proteins);果糖(Fructose);果糖磷酸类(Fructosephosphates);基因表达调控, 细菌(Gene Expression Regulation, Bacterial);质谱分析法(Mass Spectrometry);代谢组学(Metabolomics);小鼠(Mice);突变(Mutation);磷酸转移酶类(Phosphotransferases);肠炎沙门菌亚属(Salmonella enterica)

DOI 10.1038/srep28117

PMID 27403719

发布时间 2018-11-13

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A metabolic intermediate of the fructose-asparagine utilization pathway inhibits growth of a Salmonella fraB mutant.

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A metabolic intermediate of the fructose-asparagine utilization pathway inhibits growth of a Salmonella fraB mutant.

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