Critical role of RanBP2-mediated SUMOylation of Small Heterodimer Partner in maintaining bile acid homeostasis.

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作者： Dong-Hyun,Kim ^[1] ; Sanghoon,Kwon ^[1] ; Sangwon,Byun ^[1] ; Zhen,Xiao ^[2] ; Sean,Park ^[1] ; Shwu-Yuan,Wu ^[3] ; Cheng-Ming,Chiang ^[3] ; Byron,Kemper ^[1] ; Jongsook Kim,Kemper ^[1]

作者单位： Department of Molecular and Integrative Physiology, University of Illinois at Urbana-Champaign, Urbana, Illinois 61801, USA. ^[1] Laboratory of Proteomics and Analytical Technologies, Advanced Technology Program, SAIC-Frederick, Inc., Frederick National Laboratory for Cancer Research, Frederick, Maryland 21702, USA. ^[2] Simmons Comprehensive Cancer Center, Department of Biochemistry, and Department of Pharmacology, University of Texas, Southwestern Medical Center, Dallas, Texas 75390, USA. ^[3]

主题词氨基酸序列(Amino Acid Sequence);动物(Animals);胆汁酸类和盐类(Bile Acids and Salts);细胞核(Cell Nucleus);胆汁淤积(Cholestasis);疾病恶化(Disease Progression);减量调节(Down-Regulation);后成说, 遗传(Epigenesis, Genetic);Hep G2细胞(Hep G2 Cells);内环境稳定(Homeostasis);人类(Humans);肝(Liver);赖氨酸(Lysine);男(雄)性(Male);小鼠, 近交BALB C(Mice, Inbred BALB C);模型, 生物学(Models, Biological);分子伴侣(Molecular Chaperones);突变(Mutation);核孔复合蛋白质类(Nuclear Pore Complex Proteins);磷酰化(Phosphorylation);磷酸苏氨酸(Phosphothreonine);蛋白质结合(Protein Binding);蛋白质转运(Protein Transport);受体, 胞质和核(Receptors, Cytoplasmic and Nuclear);小泛素相关修饰蛋白质类(Small Ubiquitin-Related Modifier Proteins);SUMO化(Sumoylation)

DOI 10.1038/ncomms12179

PMID 27412403

发布时间 2018-11-13

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Nature communications

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Critical role of RanBP2-mediated SUMOylation of Small Heterodimer Partner in maintaining bile acid homeostasis.

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Critical role of RanBP2-mediated SUMOylation of Small Heterodimer Partner in maintaining bile acid homeostasis.

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