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A long noncoding RNA signature for ulcerative colitis identifies IFNG-AS1 as an enhancer of inflammation.

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第一作者： David,Padua

第一单位： Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California;

作者： David,Padua ^[1] ; Swapna,Mahurkar-Joshi ^[1] ; Ivy Ka Man,Law ^[1] ; Christos,Polytarchou ^[2] ; John P,Vu ^[3] ; Joseph R,Pisegna ^[3] ; David,Shih ^[4] ; Dimitrios,Iliopoulos ^[5] ; Charalabos,Pothoulakis ^[6]

作者单位： Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; ^[1] Center for Systems Biomedicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; School of Science and Technology, Nottingham Trent University, Nottingham, United Kingdom; ^[2] Division of Gastroenterology, Hepatology and Parenteral Nutrition, VA Greater Los Angeles Healthcare System and Department of Medicine David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; and. ^[3] Inflammatory Bowel and Immunobiology Research Institute, Cedars-Sinai Medical Center, Los Angeles, California. ^[4] Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; Center for Systems Biomedicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; ^[5] Division of Digestive Diseases, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, California; cpothoulakis@mednet.ucla.edu. ^[6]

关键词 inflammation inflammatory bowel disease interferon γ lncRNA ulcerative colitis