Target sequencing and CRISPR/Cas editing reveal simultaneous loss of UTX and UTY in urothelial bladder cancer.
第一作者:
Jinwoo,Ahn
第一单位:
Department of Chemistry, Yonsei University, Seoul, Korea.
作者:
主题词
细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);染色质(Chromatin);后成说, 遗传(Epigenesis, Genetic);女(雌)性(Female);基因表达谱(Gene Expression Profiling);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);HEK293细胞(HEK293 Cells);组蛋白脱甲基酶类(Histone Demethylases);人类(Humans);慢病毒属(Lentivirus);男(雄)性(Male);次要组织相容性抗原(Minor Histocompatibility Antigens);突变(Mutation);核蛋白质类(Nuclear Proteins);前瞻性研究(Prospective Studies);膀胱肿瘤(Urinary Bladder Neoplasms);X染色体失活(X Chromosome Inactivation)
DOI
10.18632/oncotarget.11207
PMID
27533081
发布时间
2019-12-10
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Oncotarget
63252-63260页
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