Alcohol promotes migration and invasion of triple-negative breast cancer cells through activation of p38 MAPK and JNK.

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作者： Ming,Zhao ^[1] ; Erin W,Howard ^[1] ; Amanda B,Parris ^[1] ; Zhiying,Guo ^[1] ; Qingxia,Zhao ^[1] ; Xiaohe,Yang ^[1]

作者单位： Department of Biology, Julius L. Chambers Biomedical/Biotechnology Research Institute, North Carolina Central University, Kannapolis, North Carolina. ^[1]

关键词 acetaldehyde alcohol cell migration/invasion oxidative stress triple-negative breast cancer

主题词醇类(Alcohols);细胞系, 肿瘤(Cell Line, Tumor);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);女(雌)性(Female);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);MAP激酶信号系统(MAP Kinase Signaling System);肿瘤侵润(Neoplasm Invasiveness);磷酰化(Phosphorylation);活性氧(Reactive Oxygen Species);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)

DOI 10.1002/mc.22538

PMID 27533114

发布时间 2022-03-16

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Molecular carcinogenesis

2017年56卷3期

849-862页

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Alcohol promotes migration and invasion of triple-negative breast cancer cells through activation of p38 MAPK and JNK.

Molecular carcinogenesis

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Alcohol promotes migration and invasion of triple-negative breast cancer cells through activation of p38 MAPK and JNK.

Molecular carcinogenesis

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Molecular carcinogenesis

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