换一批
换一批Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarction.
第一作者：
Wei,Wang
第一单位：
Department of Cardiology, Xijing Hospital, Fourth Military Medical University, Xi'an, China; and.
作者：
主题词
3-甲基-2-氧代丁酸脱氢酶(硫辛酰胺)(3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide));氨基酸类, 支链(Amino Acids, Branched-Chain);动物(Animals);印迹法, 蛋白质(Blotting, Western);冠状血管(Coronary Vessels);超声心动描记术(Echocardiography);脂肪酸类(Fatty Acids);葡萄糖(Glucose);心力衰竭(Heart Failure);原位缺口末端标记(In Situ Nick-End Labeling);结扎术(Ligation);男(雄)性(Male);小鼠(Mice);心肌梗死(Myocardial Infarction);心肌(Myocardium);利钠肽, 脑(Natriuretic Peptide, Brain);蛋白激酶类(Protein Kinases);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);心室功能障碍, 左(Ventricular Dysfunction, Left);心室重构(Ventricular Remodeling)
DOI
10.1152/ajpheart.00114.2016
PMID
27542406
发布时间
2021-12-04
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