换一批
换一批LL202 protects against dextran sulfate sodium-induced experimental colitis in mice by inhibiting MAPK/AP-1 signaling.
第一作者：
Yuan,Gao
第一单位：
State Key Laboratory of Natural Medicines, Jiangsu Key Laboratory of Carcinogenesis and Intervention, Jiangsu Key Laboratory of Drug Design and Optimization, China Pharmaceutical University, Nanjing 210009, China.
作者：
主题词
主动转运, 细胞核(Active Transport, Cell Nucleus);动物(Animals);结肠炎(Colitis);细胞因子类(Cytokines);硫酸葡聚糖(Dextran Sulfate);女(雌)性(Female);类黄酮物质(Flavonoids);基因表达调控(Gene Expression Regulation);人类(Humans);炎症(Inflammation);白细胞介素1β(Interleukin-1beta);白细胞介素6(Interleukin-6);脂多糖类(Lipopolysaccharides);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);新生血管化, 病理性(Neovascularization, Pathologic);一氧化氮合酶Ⅱ型(Nitric Oxide Synthase Type II);过氧化物酶(Peroxidase);RNA, 信使(RNA, Messenger);信号传导(Signal Transduction);转录因子AP-1(Transcription Factor AP-1);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.18632/oncotarget.11742
PMID
27590510
发布时间
2018-11-13
- 浏览5
Oncotarget
63981-63994页
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