Activation of mTOR signaling leads to orthopedic surgery-induced cognitive decline in mice through β-amyloid accumulation and tau phosphorylation.
第一作者:
Wenzhen,Shen
第一单位:
Department of Anesthesiology, Beijing Chao‑Yang Hospital, Capital Medical University, Beijing 100020, P.R. China.
作者:
主题词
淀粉样β肽类(Amyloid beta-Peptides);动物(Animals);海马(Hippocampus);男(雄)性(Male);记忆(Memory);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);矫形外科手术(Orthopedic Procedures);磷酰化(Phosphorylation);手术后并发症(Postoperative Complications);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);tau蛋白质类(tau Proteins)
DOI
10.3892/mmr.2016.5700
PMID
27599409
发布时间
2021-12-04
- 浏览0

Molecular medicine reports
3925-34页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文