Dysregulated JAK2 expression by TrkC promotes metastasis potential, and EMT program of metastatic breast cancer.

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作者： Min Soo,Kim ^[1] ; Joon,Jeong ^[2] ; Jeongbeob,Seo ^[3] ; Hae-Suk,Kim ^[4] ; Seong-Jin,Kim ^[5] ; Wook,Jin ^[1]

作者单位： Laboratory of Molecular Disease and Cell Regulation, Department of Biochemistry, School of Medicine, Gachon University, Incheon 406-840, Korea. ^[1] Department of Surgery, Gangnam Severance Hospital, Yonsei University Medical College, 712 Eonjuro, Gangnam-Gu, Seoul, 135-720, Korea. ^[2] Medicinal Chemistry, CMG Pharma, 335, CHA Bio Complex, Pangyo-ro, Bundang-gu, Seongnam-si, Gyeonggi-do, 13488, Korea. ^[3] TheragenEtex Bio Institute, TheragenEtex Co., Suwon, Gyeonggi-do 16229, Republic of Korea. ^[4] Nano-Bio Medicine Research Center, Advanced Institutes of Convergence Technology, and Department of Transdisciplinary Studies, Graduate School of Convergence Science and Technology, Seoul National University, Suwon, Kyunggi-do 16229, Republic of Korea. ^[5] Gachon Medical Research Institute, Gil Medical Center, Incheon, 405-760, Korea. ^[6]

DOI 10.1038/srep33899

PMID 27654855

发布时间 2020-07-07

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Dysregulated JAK2 expression by TrkC promotes metastasis potential, and EMT program of metastatic breast cancer.

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Dysregulated JAK2 expression by TrkC promotes metastasis potential, and EMT program of metastatic breast cancer.

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