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Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease.

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第一作者： Yasushi,Kishimoto

第一单位： Laboratory of Neurobiophysics, Kagawa School of Pharmaceutical Sciences, Tokushima Bunri University, Japan.

作者： Yasushi,Kishimoto ^[1] ; Hajime,Shishido ^[2] ; Mayumi,Sawanishi ^[1] ; Yasunori,Toyota ^[2] ; Masaki,Ueno ^[3] ; Takashi,Kubota ^[1] ; Yutaka,Kirino ^[1] ; Takashi,Tamiya ^[2] ; Nobuyuki,Kawai ^[4]

作者单位： Laboratory of Neurobiophysics, Kagawa School of Pharmaceutical Sciences, Tokushima Bunri University, Japan. ^[1] Department of Neurological Surgery, Faculty of Medicine, Kagawa University, Japan. ^[2] Department of Inflammation Pathology, Faculty of Medicine, Kagawa University, Japan. ^[3] Department of Neurological Surgery, Faculty of Medicine, Kagawa University, Japan; Department of Neurological Surgery Kagawa General Rehabilitation Hospital, Japan. ^[4]

关键词 AD, Alzheimer?s disease ANOVA, analysis of variance APP, amyloid precursor protein Amyloid precursor protein CR, conditioned response CS, conditioned stimulus Eyeblink conditioning Rotarod test Spontaneous physical activity TBI, traumatic brain injury Traumatic brain injury US, unconditioned stimulus

DOI 10.1016/j.dib.2016.08.041

PMID 27656663

发布时间 2020-10-01

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Data in brief

2016年9卷

62-7页

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Data in brief

2016年9卷

62-7页

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Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease.

Data in brief

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Data in brief

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Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease.

Data in brief

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Data in brief

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