Bacterial Abscess Formation Is Controlled by the Stringent Stress Response and Can Be Targeted Therapeutically.

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作者： Sarah C,Mansour ^[1] ; Daniel,Pletzer ^[1] ; César,de la Fuente-Núñez ^[1] ; Paul,Kim ^[1] ; Gordon Y C,Cheung ^[2] ; Hwang-Soo,Joo ^[2] ; Michael,Otto ^[2] ; Robert E W,Hancock ^[3]

作者单位： Centre for Microbial Diseases and Immunity Research, Department of Microbiology and Immunology, University of British Columbia, Vancouver, British Columbia V6T 1Z4, Canada. ^[1] Pathogen Molecular Genetics Section, Laboratory of Bacteriology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20814, United States. ^[2] Centre for Microbial Diseases and Immunity Research, Department of Microbiology and Immunology, University of British Columbia, Vancouver, British Columbia V6T 1Z4, Canada. Electronic address: bob@hancocklab.com. ^[3]

关键词 Cationic peptide DJK-5 Pseudomonas aeruginosa Staphylococcus aureus ppGpp

主题词脓肿(Abscess);动物(Animals);抗菌药(Anti-Bacterial Agents);抗微生物阳离子肽类(Antimicrobial Cationic Peptides);细菌毒素类(Bacterial Toxins);生物膜(Biofilms);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);耐甲氧西林金黄色葡萄球菌(Methicillin-Resistant Staphylococcus aureus);小鼠(Mice);微生物敏感性试验(Microbial Sensitivity Tests);葡萄球菌感染(Staphylococcal Infections);金黄色葡萄球菌(Staphylococcus aureus);应激, 生理学(Stress, Physiological);泛素硫酯酶(Ubiquitin Thiolesterase)

DOI 10.1016/j.ebiom.2016.09.015

PMID 27658736

发布时间 2020-12-09

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EBioMedicine

2016年12卷

219-226页

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Bacterial Abscess Formation Is Controlled by the Stringent Stress Response and Can Be Targeted Therapeutically.

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