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Endothelial to mesenchymal transition contributes to arsenic-trioxide-induced cardiac fibrosis.

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第一作者: Yong,Zhang
第一单位: Department of Pharmacology (the State-Province Key Laboratories of Biomedicine-Pharmaceutics of China, Key Laboratory of Cardiovascular Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, 150081, China.;Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Harbin, 150086, China.
作者单位: Department of Pharmacology (the State-Province Key Laboratories of Biomedicine-Pharmaceutics of China, Key Laboratory of Cardiovascular Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, 150081, China.;Institute of Metabolic Disease, Heilongjiang Academy of Medical Science, Harbin, 150086, China. [1] Department of Pharmacology (the State-Province Key Laboratories of Biomedicine-Pharmaceutics of China, Key Laboratory of Cardiovascular Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, 150081, China. [2] Center for Endemic Disease Control, Chinese Center for Disease Control and Prevention, Key Lab of Etiology and Epidemiology, Education Bureau of Heilongjiang Province &Ministry of Health (23618504), Harbin Medical University, Harbin, 150081, China. [3] The Research Center, Sechenov First Moscow State Medical University, Moscow, 119991, Russia. [4] Department of preventive and emergency cardiology, Sechenov First Moscow State Medical University, Moscow, 119991, Russia. [5] Department of Pharmacology (the State-Province Key Laboratories of Biomedicine-Pharmaceutics of China, Key Laboratory of Cardiovascular Research, Ministry of Education), College of Pharmacy, Harbin Medical University, Harbin, 150081, China.;Department of Pharmacology and Therapeutics, Melbourne School of Biomedical Sciences, Faculty of Medicine, Dentistry and Health Sciences, The University of Melbourne, Melbourne, 3010, Australia. [6]
DOI 10.1038/srep33787
PMID 27671604
发布时间 2019-11-20
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