TAK1 knockdown enhances lipopolysaccharide-induced secretion of proinflammatory cytokines in myeloid cells via unleashing MEKK3 activity.
第一作者:
Kun,Li
第一单位:
Institute of Immunology, College of Life Science & Technology, Dalian University, Dalian, Liaoning, China; College of Medicine, Dalian University, Dalian, Liaoning, China.
作者:
主题词
动物(Animals);细胞分化(Cell Differentiation);细胞因子类(Cytokines);人类(Humans);免疫, 先天(Immunity, Innate);炎症(Inflammation);炎症介导素类(Inflammation Mediators);脂多糖类(Lipopolysaccharides);MAP激酶激酶激酶3(MAP Kinase Kinase Kinase 3);MAP激酶激酶激酶类(MAP Kinase Kinase Kinases);巨噬细胞活化(Macrophage Activation);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 近交系(Mice, Inbred Strains);NF-κB(NF-kappa B);RNA, 小分子干扰(RNA, Small Interfering)
DOI
10.1016/j.cellimm.2016.09.011
PMID
27671672
发布时间
2018-08-14
- 浏览6
Cellular immunology
193-198页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文