Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.
第一作者:
Tahereh,Javaheri
第一单位:
Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria.;Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria.
作者:
医学主题词
动物(Animals);动物, 新生(Animals, Newborn);细胞凋亡(Apoptosis);骨和骨组织(Bone and Bones);细胞周期(Cell Cycle);细胞分化(Cell Differentiation);细胞增殖(Cell Proliferation);细胞存活(Cell Survival);细胞转化, 肿瘤(Cell Transformation, Neoplastic);四肢(Extremities);基因表达谱(Gene Expression Profiling);基因敲低技术(Gene Knockdown Techniques);人类(Humans);间质干细胞(Mesenchymal Stem Cells);小鼠(Mice);癌基因蛋白质类, 融合(Oncogene Proteins, Fusion);骨生成(Osteogenesis);原癌基因蛋白质c-fli-1(Proto-Oncogene Protein c-fli-1);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);RNA结合蛋白质EWS(RNA-Binding Protein EWS);信号传导(Signal Transduction);转导, 遗传(Transduction, Genetic)
DOI
10.1038/cddis.2016.268
PMID
27735950
发布时间
2025-05-30
- 浏览2
Cell death & disease
e2419页
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