ISG12a inhibits HCV replication and potentiates the anti-HCV activity of IFN-α through activation of the Jak/STAT signaling pathway independent of autophagy and apoptosis.

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作者： Yanzhao,Chen ^[1] ; Baihai,Jiao ^[1] ; Min,Yao ^[1] ; Xuezhen,Shi ^[1] ; Zhebin,Zheng ^[2] ; Shilin,Li ^[3] ; Limin,Chen ^[4]

作者单位： Institute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical College, Provincial Key Laboratory for Transfusion-transmitted Infectious Diseases of Sichuan Province, Chengdu, Sichuan, 610052 China. ^[1] Chengdu University, Sichuan Industrial Institute of Antibiotics, Antibiotics Research and Re-evaluation Key Laboratory of Sichuan Province, China. ^[2] Institute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical College, Provincial Key Laboratory for Transfusion-transmitted Infectious Diseases of Sichuan Province, Chengdu, Sichuan, 610052 China. Electronic address: shilin-li@hotmail.com. ^[3] Institute of Blood Transfusion, Chinese Academy of Medical Sciences and Peking Union Medical College, Provincial Key Laboratory for Transfusion-transmitted Infectious Diseases of Sichuan Province, Chengdu, Sichuan, 610052 China; Toronto General Research Institute, University of Toronto, Toronto, ON M5G 1L6 Canada. Electronic address: Limin_chen_99@yahoo.com. ^[4]

关键词 Apoptosis Autophagy HCV IFN-α ISG12a Jak/STAT

主题词细胞凋亡(Apoptosis);自噬(Autophagy);细胞系(Cell Line);细胞, 培养的(Cells, Cultured);基因表达(Gene Expression);肝炎病毒属(Hepacivirus);丙型肝炎(Hepatitis C);人类(Humans);干扰素Ⅰ型(Interferon Type I);干扰素α(Interferon-alpha);干扰素β(Interferon-beta);Janus激酶类(Janus Kinases);膜蛋白质类(Membrane Proteins);STAT转录因子类(STAT Transcription Factors);信号传导(Signal Transduction);病毒复制(Virus Replication)

DOI 10.1016/j.virusres.2016.10.013

PMID 27777077

发布时间 2017-12-18

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Virus research

2017年227卷

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ISG12a inhibits HCV replication and potentiates the anti-HCV activity of IFN-α through activation of the Jak/STAT signaling pathway independent of autophagy and apoptosis.

Virus research

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Virus research

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ISG12a inhibits HCV replication and potentiates the anti-HCV activity of IFN-α through activation of the Jak/STAT signaling pathway independent of autophagy and apoptosis.

Virus research

相似文献

Virus research

相关期刊

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