In vivo Patterns of Tau Pathology, Amyloid-β Burden, and Neuronal Dysfunction in Clinical Variants of Alzheimer's Disease.

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作者： Julian,Dronse ^[1] ; Klaus,Fliessbach ^[2] ; Gérard N,Bischof ^[3] ; Boris,von Reutern ^[4] ; Jennifer,Faber ^[2] ; Jochen,Hammes ^[5] ; Georg,Kuhnert ^[1] ; Bernd,Neumaier ^[2] ; Oezguer A,Onur ^[4] ; Juraj,Kukolja ^[6] ; Thilo,van Eimeren ^[5] ; Frank,Jessen ^[5] ; Gereon R,Fink ^[7] ; Thomas,Klockgether ^[8] ; Alexander,Drzezga ^[1]

作者单位： Department of Neurology, University Hospital Cologne, Cologne, Germany. ^[1] Cognitive Neuroscience, Institute of Neuroscience and Medicine (INM-3), Research Center Jülich, Jülich, Germany. ^[2] Department of Psychiatry and Psychotherapy, University Hospital Bonn, Bonn, Germany. ^[3] German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany. ^[4] Multimodal Neuroimaging Group, Department of Nuclear Medicine, University Hospital Cologne, Cologne, Germany. ^[5] Department of Neurology, University Hospital Bonn, Bonn, Germany. ^[6] Nuclear Chemistry, Institute of Neuroscience and Medicine (INM-5), Research Center Jülich, Jülich, Germany. ^[7] Institute of Radiochemistry and Experimental Molecular Imaging, University Hospital Cologne, Cologne, Germany. ^[8] Department of Psychiatry, University Hospital Cologne, Cologne, Germany. ^[9]

关键词 18F-AV-1451 18F-FDG Alzheimer’s disease Pittsburghcompound B T-807 amyloid molecular imaging multimodal imaging positron-emission tomography tau protein