Genetic and pharmacological inhibition of CDK9 drives neutrophil apoptosis to resolve inflammation in zebrafish in vivo.

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第一作者： Laura J,Hoodless

第一单位： MRC Centre for Inflammation Research, The Queen's Medical Research Institute, The University of Edinburgh, Edinburgh, EH16 4TJ, United Kingdom.

作者： Laura J,Hoodless ^[1] ; Christopher D,Lucas ^[1] ; Rodger,Duffin ^[1] ; Martin A,Denvir ^[2] ; Christopher,Haslett ^[1] ; Carl S,Tucker ^[2] ; Adriano G,Rossi ^[1]

作者单位： MRC Centre for Inflammation Research, The Queen's Medical Research Institute, The University of Edinburgh, Edinburgh, EH16 4TJ, United Kingdom. ^[1] BHF Centre for Cardiovascular Science, The Queen's Medical Research Institute, The University of Edinburgh, Edinburgh, EH16 4TJ, United Kingdom. ^[2]

主题词动物(Animals);细胞凋亡(Apoptosis);细胞周期蛋白依赖激酶9(Cyclin-Dependent Kinase 9);疾病模型, 动物(Disease Models, Animal);类黄酮物质(Flavonoids);基因敲低技术(Gene Knockdown Techniques);炎症(Inflammation);巨噬细胞(Macrophages);中性白细胞(Neutrophils);哌啶类(Piperidines);蛋白激酶抑制剂(Protein Kinase Inhibitors);吡唑类(Pyrazoles);核糖核蛋白类(Ribonucleoproteins);斑马鱼(Zebrafish);斑马鱼蛋白质类(Zebrafish Proteins)

DOI 10.1038/srep36980

PMID 27833165

发布时间 2024-03-26

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Genetic and pharmacological inhibition of CDK9 drives neutrophil apoptosis to resolve inflammation in zebrafish in vivo.

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Genetic and pharmacological inhibition of CDK9 drives neutrophil apoptosis to resolve inflammation in zebrafish in vivo.

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