换一批Gankyrin sustains PI3K/GSK-3β/β-catenin signal activation and promotes colorectal cancer aggressiveness and progression.
第一作者：
Feng,He
第一单位：
Department of General Surgery, Guangzhou Digestive Disease Center, Guangzhou First People's Hospital, Guangzhou Medical University, Guangzhou 510180, China.;Department of Pathology, Interdepartmental Program in Vascular Biology and Therapeutics, Yale University School of Medicine, New Haven, Connecticut 06520, USA.
作者：
医学主题词
动物(Animals);抗代谢药, 抗肿瘤(Antimetabolites, Antineoplastic);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);结直肠肿瘤(Colorectal Neoplasms);计算生物学(Computational Biology);数据库, 遗传学(Databases, Genetic);抗药性, 肿瘤(Drug Resistance, Neoplasm);女(雌)性(Female);氟尿嘧啶(Fluorouracil);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);HCT116细胞(HCT116 Cells);人类(Humans);男(雄)性(Male);小鼠, 近交BALB C(Mice, Inbred BALB C);中年人(Middle Aged);肿瘤侵润(Neoplasm Invasiveness);新生血管化, 病理性(Neovascularization, Pathologic);表型(Phenotype);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);RNA干扰(RNA Interference);时间因素(Time Factors);转染(Transfection);β连环素(beta Catenin)
DOI
10.18632/oncotarget.13215
PMID
27835604
发布时间
2018-11-13
- 浏览16
Oncotarget
81156-81171页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
