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首页> Neuropharmacology> Pharmacological inhibition of PTEN attenuates cognitive deficits caused by neonatal repeated exposures to isoflurane via inhibition of NR2B-mediated tau phosphorylation in rats.

Pharmacological inhibition of PTEN attenuates cognitive deficits caused by neonatal repeated exposures to isoflurane via inhibition of NR2B-mediated tau phosphorylation in rats.

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作者： Lei,Tan ^[1] ; Xin,Chen ^[2] ; Wei,Wang ^[3] ; Jianfang,Zhang ^[1] ; Shiyong,Li ^[1] ; Yilin,Zhao ^[1] ; Jintao,Wang ^[1] ; Ailin,Luo ^[4]

作者单位： Department of Anesthesiology, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China. ^[1] Department of Anesthesiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China. ^[2] Department of Anesthesiology, First Hospital of Xian Jiaotong University, Xi'an 710061, China. ^[3] Department of Anesthesiology, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China. Electronic address: alluo@tjh.tjmu.edu.cn. ^[4]

关键词 Developing brain Isoflurane NR2B-containing NMDAR Phosphatase and tensin homolog deleted on chromosome 10 Tau phosphorylation

主题词动物(Animals);动物, 新生(Animals, Newborn);海马(Hippocampus);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);异氟醚(Isoflurane);男(雄)性(Male);迷宫学习(Maze Learning);膜蛋白质类(Membrane Proteins);PTEN磷酸水解酶(PTEN Phosphohydrolase);磷酰化(Phosphorylation);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);突触(Synapses);突触蛋白类(Synapsins);突触囊泡蛋白(Synaptophysin);突触体相关蛋白质25(Synaptosomal-Associated Protein 25);tau蛋白质类(tau Proteins)

DOI 10.1016/j.neuropharm.2016.11.008

PMID 27836791

发布时间 2018-09-12

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Neuropharmacology

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