Qki deficiency maintains stemness of glioma stem cells in suboptimal environment by downregulating endolysosomal degradation.

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作者： Takashi,Shingu ^[1] ; Allen L,Ho ^[2] ; Liang,Yuan ^[1] ; Xin,Zhou ^[1] ; Congxin,Dai ^[1] ; Siyuan,Zheng ^[3] ; Qianghu,Wang ^[4] ; Yi,Zhong ^[3] ; Qing,Chang ^[4] ; James W,Horner ^[5] ; Brandon D,Liebelt ^[6] ; Yu,Yao ^[6] ; Baoli,Hu ^[7] ; Yiwen,Chen ^[8] ; Gregory N,Fuller ^[9] ; Roeland G W,Verhaak ^[1] ; Amy B,Heimberger ^[4] ; Jian,Hu ^[10]

作者单位： Department of Cancer Biology, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[1] Department of Neurosurgery, Stanford University, Stanford, California, USA. ^[2] Department of Genomic Medicine, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[3] Department of Bioinformatics and Computational Biology, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[4] Department of Epigenetics and Molecular Carcinogenesis, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[5] Institute for Applied Cancer Science, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[6] Department of Neurosurgery, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[7] Department of Neurosurgery, Houston Methodist Neurological Institute, Houston, Texas, USA. ^[8] Department of Neurosurgery, Fudan University Huashan Hospital, Shanghai, China. ^[9] Department of Pathology, University of Texas MD Anderson Cancer Center, Houston, Texas, USA. ^[10]