Forward Genetic Screen in <i>Caenorhabditis elegans</i> Suggests F57A10.2 and acp-4 As Suppressors of C9ORF72 Related Phenotypes.

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作者： Xin,Wang ^[1] ; Limin,Hao ^[2] ; Taixiang,Saur ^[2] ; Katelyn,Joyal ^[2] ; Ying,Zhao ^[3] ; Desheng,Zhai ^[2] ; Jie,Li ^[4] ; Mochtar,Pribadi ^[5] ; Giovanni,Coppola ^[5] ; Bruce M,Cohen ^[2] ; Edgar A,Buttner ^[2]

作者单位： School of Public Health, Xinxiang Medical UniversityXinxiang, China; Program for Neuropsychiatric Research, McLean Hospital and Harvard Medical SchoolBelmont, MA, USA. ^[1] Program for Neuropsychiatric Research, McLean Hospital and Harvard Medical School Belmont, MA, USA. ^[2] School of Pharmacy, Xinxiang Medical University Xinxiang, China. ^[3] Tianjin Mental Health Center Tianjin, China. ^[4] Semel Institute for Neuroscience and Human Behavior, David Geffen School of Medicine, University of California, Los Angeles Los Angeles, CA, USA. ^[5]

关键词 ACP2 ALS-FTLD C9ORF72 Caenorhabditis elegans VAPB amyotrophic lateral sclerosis

DOI 10.3389/fnmol.2016.00113

PMID 27877110

发布时间 2020-09-30

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Frontiers in molecular neuroscience

2016年9卷

113页

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Forward Genetic Screen in <i>Caenorhabditis elegans</i> Suggests F57A10.2 and acp-4 As Suppressors of C9ORF72 Related Phenotypes.

Frontiers in molecular neuroscience

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Frontiers in molecular neuroscience

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Forward Genetic Screen in <i>Caenorhabditis elegans</i> Suggests F57A10.2 and acp-4 As Suppressors of C9ORF72 Related Phenotypes.

Frontiers in molecular neuroscience

相似文献

Frontiers in molecular neuroscience

相关期刊

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