Upregulation of FAM83D promotes malignant phenotypes of lung adenocarcinoma by regulating cell cycle.

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作者： Run,Shi ^[1] ; Jing,Sun ^[2] ; Qi,Sun ^[3] ; Quanli,Zhang ^[4] ; Wenjie,Xia ^[1] ; Gaochao,Dong ^[5] ; Anpeng,Wang ^[1] ; Feng,Jiang ^[5] ; Lin,Xu ^[5]

作者单位： Jiangsu Key Laboratory of Molecular and Translational Cancer Research, Cancer Institute of Jiangsu ProvinceJiangsu, China; Department of Thoracic Surgery, Affiliated Cancer Hospital, Nanjing Medical UniversityJiangsu, China; The Fourth Clinical College of Nanjing Medical UniversityNanjing, China. ^[1] The First Clinical College of Nanjing Medical University Nanjing, China. ^[2] Department of Cardiothoracic Surgery at Jinling Hospital, Southern Medical University Jinling, China. ^[3] Jiangsu Key Laboratory of Molecular and Translational Cancer Research, Cancer Institute of Jiangsu Province Jiangsu, China. ^[4] Jiangsu Key Laboratory of Molecular and Translational Cancer Research, Cancer Institute of Jiangsu ProvinceJiangsu, China; Department of Thoracic Surgery, Affiliated Cancer Hospital, Nanjing Medical UniversityJiangsu, China. ^[5]

关键词 FAM83D TCGA cell cycle lung adenocarcinoma

PMID 27904773

发布时间 2020-09-30

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American journal of cancer research

2016年6卷11期

2587-2598页

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Upregulation of FAM83D promotes malignant phenotypes of lung adenocarcinoma by regulating cell cycle.

American journal of cancer research

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American journal of cancer research

相关期刊

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Upregulation of FAM83D promotes malignant phenotypes of lung adenocarcinoma by regulating cell cycle.

American journal of cancer research

相似文献

American journal of cancer research

相关期刊

检索历史清除