换一批Renin-angiotensin system transgenic mouse model recapitulates pathophysiology similar to human preeclampsia with renal injury that may be mediated through VEGF.
第一作者：
J Morgan,Denney
第一单位：
Department of Obstetrics & Gynecology, Division of Maternal-Fetal Medicine, University of Wisconsin School of Medicine and Public Health-Madison, Madison, Wisconsin.
作者：
医学主题词
白蛋白尿(Albuminuria);血管紧张素原(Angiotensinogen);动物(Animals);血压(Blood Pressure);疾病模型, 动物(Disease Models, Animal);内皮细胞(Endothelial Cells);女(雌)性(Female);疾病遗传易感性(Genetic Predisposition to Disease);孕龄(Gestational Age);人类(Humans);肾疾病(Kidney Diseases);肾小球(Kidney Glomerulus);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);表型(Phenotype);胎盘(Placenta);先兆子痫(Pre-Eclampsia);妊娠(Pregnancy);肾素(Renin);肾素-血管紧张素系统(Renin-Angiotensin System);信号传导(Signal Transduction);血管内皮生长因子A(Vascular Endothelial Growth Factor A);血管内皮生长因子受体1(Vascular Endothelial Growth Factor Receptor-1);血管收缩(Vasoconstriction);血管舒张(Vasodilation)
DOI
10.1152/ajprenal.00108.2016
PMID
27927648
发布时间
2018-06-06
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