Prenatal alcohol exposure potentiates chronic neuropathic pain, spinal glial and immune cell activation and alters sciatic nerve and DRG cytokine levels.

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作者： Shahani,Noor ^[1] ; Joshua J,Sanchez ^[1] ; Arden G,Vanderwall ^[2] ; Melody S,Sun ^[1] ; Jessie R,Maxwell ^[3] ; Suzy,Davies ^[1] ; Lauren L,Jantzie ^[4] ; Timothy R,Petersen ^[5] ; Daniel D,Savage ^[4] ; Erin D,Milligan ^[6]

作者单位： Department of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. ^[1] Department of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA; Department of Anesthesiology and Critical Care Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. ^[2] Department of Pediatrics, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. ^[3] Department of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA; Department of Pediatrics, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. ^[4] Department of Anesthesiology and Critical Care Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. ^[5] Department of Neurosciences, School of Medicine, University of New Mexico Health Sciences Center, Albuquerque, NM 87131-0001, USA. Electronic address: EMilligan@salud.unm.edu. ^[6]

关键词 Astrocytes Chronic constriction injury Fetal alcohol spectrum disorder Inflammatory cytokines Microglia Neuroimmune interactions Neuropathic pain Prenatal alcohol exposure Sciatic nerve Spinal glia β-Integrin adhesion molecules