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首页> The Journal of biological chemistry> Histone H1 and Chromosomal Protein HMGN2 Regulate Prolactin-induced STAT5 Transcription Factor Recruitment and Function in Breast Cancer Cells.

Histone H1 and Chromosomal Protein HMGN2 Regulate Prolactin-induced STAT5 Transcription Factor Recruitment and Function in Breast Cancer Cells.

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作者： Suzanne M,Schauwecker ^[1] ; J Julie,Kim ^[2] ; Jonathan D,Licht ^[3] ; Charles V,Clevenger ^[4]

作者单位： From the Department of Pathology and. ^[1] the Division of Reproductive Science in Medicine, Department of Obstetrics and Gynecology, Robert H. Lurie Comprehensive Cancer Center, Feinberg School of Medicine, Northwestern University, Chicago, Illinois 60611. ^[2] the Division of Hematology and Oncology, Department of Medicine, University of Florida Health Cancer Center, Gainesville, Florida 32610, and. ^[3] the Department of Pathology, Virginia Commonwealth University, Richmond, Virginia 23298 charles.clevenger@vcuhealth.org. ^[4]

关键词 STAT transcription factor breast cancer chromatin remodeling linker histone H1 non-histone chromosomal protein HMG-17 (HMGN2)prolactin prolactin receptor signal transducers and activators of transcription 5 (STAT5)transcription regulation

主题词乳腺肿瘤(Breast Neoplasms);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);女(雌)性(Female);HMGN2蛋白质(HMGN2 Protein);组蛋白类(Histones);人类(Humans);催乳素(Prolactin);蛋白质加工, 转译后(Protein Processing, Post-Translational);STAT5转录因子(STAT5 Transcription Factor);转录, 遗传(Transcription, Genetic)

DOI 10.1074/jbc.M116.764233

PMID 28035005

发布时间 2021-02-05

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The Journal of biological chemistry

The Journal of biological chemistry

2017年292卷6期

2237-2254页

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