Glucocorticoids curtail stimuli-induced CREB phosphorylation in TRH neurons through interaction of the glucocorticoid receptor with the catalytic subunit of protein kinase A.
第一作者:
Israim,Sotelo-Rivera
第一单位:
Departamento de Genética del Desarrollo y Fisiología Molecular, Instituto de Biotecnología, Universidad Nacional Autónoma de México (UNAM), UNAM, A.P. 510-3, Cuernavaca, Morelos, 62271, Mexico.
作者:
主题词
动物(Animals);细胞系, 肿瘤(Cell Line, Tumor);细胞, 培养的(Cells, Cultured);冷温度(Cold Temperature);cAMP反应元件结合蛋白质(Cyclic AMP Response Element-Binding Protein);环AMP依赖性蛋白激酶类(Cyclic AMP-Dependent Protein Kinases);地塞米松(Dexamethasone);糖皮质激素类(Glucocorticoids);下丘脑(Hypothalamus);神经元(Neurons);下丘脑室旁核(Paraventricular Hypothalamic Nucleus);磷酰化(Phosphorylation);大鼠(Rats);大鼠, Wistar(Rats, Wistar);受体, 糖皮质激素(Receptors, Glucocorticoid);促甲状腺素释放激素(Thyrotropin-Releasing Hormone)
DOI
10.1007/s12020-016-1223-z
PMID
28063130
发布时间
2018-11-13
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Endocrine
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