Quercetin induces apoptosis and autophagy in primary effusion lymphoma cells by inhibiting PI3K/AKT/mTOR and STAT3 signaling pathways.
第一作者:
Marisa,Granato
第一单位:
Department of Experimental Medicine, "Sapienza" University of Rome, Italy.
作者:
主题词
抗肿瘤药(Antineoplastic Agents);抗肿瘤药, 植物(Antineoplastic Agents, Phytogenic);细胞凋亡(Apoptosis);自噬(Autophagy);B-淋巴细胞(B-Lymphocytes);细胞系, 肿瘤(Cell Line, Tumor);细胞, 培养的(Cells, Cultured);减量调节(Down-Regulation);药物激动作用(Drug Agonism);人类(Humans);白细胞介素类(Interleukins);淋巴瘤, 原发性渗出性(Lymphoma, Primary Effusion);肿瘤蛋白质类(Neoplasm Proteins);磷酸肌醇3-激酶(Phosphatidylinositol 3-Kinase);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);槲皮素(Quercetin);重组融合蛋白质类(Recombinant Fusion Proteins);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases)
DOI
10.1016/j.jnutbio.2016.12.011
PMID
28092744
发布时间
2021-12-04
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