换一批SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.
第一作者：
Tae Yun,Kim
第一单位：
Department of Medicine, Division of Cardiology, Cardiovascular Research Center, Rhode Island Hospital, The Warren Alpert Medical School of Brown University, 1 Hoppin Street, Providence, RI, 02903-4141, USA.
作者：
医学主题词
动物(Animals);心律失常, 心性(Arrhythmias, Cardiac);钙信号(Calcium Signaling);心脏扩大(Cardiomegaly);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);吲哚类(Indoles);动力学(Kinetics);膜电位, 线粒体(Membrane Potential, Mitochondrial);线粒体, 心脏(Mitochondria, Heart);肌细胞, 心脏(Myocytes, Cardiac);氧化还原(Oxidation-Reduction);肟类(Oximes);吡唑类(Pyrazoles);嘧啶类(Pyrimidines);大鼠(Rats);活性氧(Reactive Oxygen Species);兰尼碱受体钙释放通道(Ryanodine Receptor Calcium Release Channel);肌浆网(Sarcoplasmic Reticulum);小电导钙激活钾通道(Small-Conductance Calcium-Activated Potassium Channels)
DOI
10.1093/cvr/cvx005
PMID
28096168
发布时间
2020-08-27
- 浏览8
Cardiovascular research
343-353页
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