Alcoholic fatty liver is enhanced in CYP2A5 knockout mice: The role of the PPARα-FGF21 axis.

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第一作者： Xue,Chen

第一单位： Department of Health Sciences, College of Public Health, East Tennessee State University, Johnson City, TN, United States.

作者： Xue,Chen ^[1] ; Stephen C,Ward ^[2] ; Arthur I,Cederbaum ^[3] ; Huabao,Xiong ^[4] ; Yongke,Lu ^[5]

作者单位： Department of Health Sciences, College of Public Health, East Tennessee State University, Johnson City, TN, United States. ^[1] Department of Pathology, Icahn School of Medicine at Mount Sinai, New York, New York, United States. ^[2] Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, New York, United States. ^[3] Division of Immunology, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, New York, United States. ^[4] Department of Health Sciences, College of Public Health, East Tennessee State University, Johnson City, TN, United States; Department of Pharmacological Sciences, Icahn School of Medicine at Mount Sinai, New York, New York, United States. Electronic address: luy004@etsu.edu. ^[5]

关键词 Alcoholic fatty liver CYP2A5 FGF21 PPARα

主题词动物(Animals);芳基烃羟化酶类(Aryl Hydrocarbon Hydroxylases);脂肪肝, 酒精性(Fatty Liver, Alcoholic);女(雌)性(Female);成纤维细胞生长因子(Fibroblast Growth Factors);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);NF-E2相关因子2(NF-E2-Related Factor 2);PPARα(PPAR alpha)

DOI 10.1016/j.tox.2017.01.016

PMID 28131861

发布时间 2018-11-13

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Toxicology

2017年379卷

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Alcoholic fatty liver is enhanced in CYP2A5 knockout mice: The role of the PPARα-FGF21 axis.

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Alcoholic fatty liver is enhanced in CYP2A5 knockout mice: The role of the PPARα-FGF21 axis.

Toxicology

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Toxicology

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