Racemic Salsolinol and its Enantiomers Act as Agonists of the μ-Opioid Receptor by Activating the Gi Protein-Adenylate Cyclase Pathway.

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作者： Pablo,Berríos-Cárcamo ^[1] ; María E,Quintanilla ^[2] ; Mario,Herrera-Marschitz ^[2] ; Vasilis,Vasiliou ^[3] ; Gerald,Zapata-Torres ^[4] ; Mario,Rivera-Meza ^[5]

作者单位： Program of Molecular and Clinical Pharmacology, Faculty of Medicine, Institute of Biomedical Sciences, University of ChileSantiago, Chile; Department of Environmental Health Sciences, Yale School of Public HealthNew Haven, CT, USA. ^[1] Program of Molecular and Clinical Pharmacology, Faculty of Medicine, Institute of Biomedical Sciences, University of Chile Santiago, Chile. ^[2] Department of Environmental Health Sciences, Yale School of Public Health New Haven, CT, USA. ^[3] Department of Analytical and Inorganic Chemistry, Faculty of Chemical and Pharmaceutical Sciences, University of Chile Santiago, Chile. ^[4] Department of Pharmacological and Toxicological Chemistry, Faculty of Chemical and Pharmaceutical Sciences, University of Chile Santiago, Chile. ^[5]

关键词 molecular docking naltrexone salsolinol β-arrestin μ-opioid receptor

DOI 10.3389/fnbeh.2016.00253

PMID 28167903

发布时间 2020-10-01

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Frontiers in behavioral neuroscience

2016年10卷

253页

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Racemic Salsolinol and its Enantiomers Act as Agonists of the μ-Opioid Receptor by Activating the Gi Protein-Adenylate Cyclase Pathway.

Frontiers in behavioral neuroscience

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Frontiers in behavioral neuroscience

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Racemic Salsolinol and its Enantiomers Act as Agonists of the μ-Opioid Receptor by Activating the Gi Protein-Adenylate Cyclase Pathway.

Frontiers in behavioral neuroscience

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Frontiers in behavioral neuroscience

相关期刊

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