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首页> PloS one> Association of IFIH1 and pro-inflammatory mediators: Potential new clues in SLE-associated pathogenesis.

Association of IFIH1 and pro-inflammatory mediators: Potential new clues in SLE-associated pathogenesis.

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作者： Melissa E,Munroe ^[1] ; Nathan,Pezant ^[1] ; Michael A,Brown ^[2] ; Dustin A,Fife ^[1] ; Joel M,Guthridge ^[1] ; Jennifer A,Kelly ^[1] ; Graham,Wiley ^[1] ; Patrick M,Gaffney ^[1] ; Judith A,James ^[3] ; Courtney G,Montgomery ^[2]

作者单位： Arthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, OK, United States of America. ^[1] Arthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, OK, United States of America.;Department of Biostatistics and Epidemiology, University of Oklahoma Health Sciences Center, Oklahoma City, OK, United States of America. ^[2] Arthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, OK, United States of America.;Department of Medicine and Pathology, University of Oklahoma Health Sciences Center, Oklahoma City, OK, United States of America. ^[3]

主题词成年人(Adult);老年人(Aged);动物(Animals);趋化因子CXCL10(Chemokine CXCL10);遗传关联研究(Genetic Association Studies);疾病遗传易感性(Genetic Predisposition to Disease);基因型(Genotype);人类(Humans);炎症介导素类(Inflammation Mediators);干扰素β(Interferon-beta);白细胞介素6(Interleukin-6);红斑狼疮, 系统性(Lupus Erythematosus, Systemic);小鼠(Mice);中年人(Middle Aged);多态性, 单核苷酸(Polymorphism, Single Nucleotide);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)

DOI 10.1371/journal.pone.0171193

PMID 28234905

发布时间 2024-09-22

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PloS one

PloS one

2017年12卷2期

e0171193页

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