Cytotoxicity of Natural Killer Cells Activated Through NKG2D Contributes to the Development of Bronchiolitis Obliterans in a Murine Heterotopic Tracheal Transplant Model.
第一作者:
T,Kawakami
第一单位:
Department of Immunobiology, Institute of Development, Aging and Cancer, Tohoku University, Aoba-ku, Sendai, Miyagi, Japan.;Department of Thoracic Surgery, Graduate School of Medicine, Tohoku University, Aoba-ku, Sendai, Miyagi, Japan.
作者:
医学主题词
动物(Animals);闭塞性细支气管炎(Bronchiolitis Obliterans);CD8阳性T淋巴细胞(CD8-Positive T-Lymphocytes);细胞, 培养的(Cells, Cultured);疾病模型, 动物(Disease Models, Animal);移植物排斥(Graft Rejection);同源盒结构域蛋白质类(Homeodomain Proteins);免疫, 细胞(Immunity, Cellular);杀伤细胞, 天然(Killer Cells, Natural);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, SCID(Mice, SCID);NK细胞凝集素样受体亚家族K(NK Cell Lectin-Like Receptor Subfamily K);气管(Trachea);移植, 异位(Transplantation, Heterotopic)
DOI
10.1111/ajt.14257
PMID
28251796
发布时间
2023-01-24
- 浏览12
American journal of transplantation
2338-2349页
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