换一批
换一批Reduced dosage of β-catenin provides significant rescue of cardiac outflow tract anomalies in a Tbx1 conditional null mouse model of 22q11.2 deletion syndrome.
第一作者：
Silvia E,Racedo
第一单位：
Department of Genetics, Albert Einstein College of Medicine, Bronx, NY, United States of America.
作者：
主题词
动物(Animals);细胞凋亡(Apoptosis);心血管畸形(Cardiovascular Abnormalities);细胞分化(Cell Differentiation);细胞增殖(Cell Proliferation);DiGeorge综合征(DiGeorge Syndrome);疾病模型, 动物(Disease Models, Animal);基因表达谱(Gene Expression Profiling);基因表达调控, 发育期(Gene Expression Regulation, Developmental);人类(Humans);原位杂交(In Situ Hybridization);中胚层(Mesoderm);小鼠, 基因敲除(Mice, Knockout);小鼠, 转基因(Mice, Transgenic);显微镜检查, 荧光(Microscopy, Fluorescence);肌细胞, 心脏(Myocytes, Cardiac);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);T盒域蛋白质类(T-Box Domain Proteins);动脉干(Truncus Arteriosus);β连环素(beta Catenin)
DOI
10.1371/journal.pgen.1006687
PMID
28346476
发布时间
2019-02-02
- 浏览8
PLoS genetics
e1006687页
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