Altered Ca<sup>2+</sup> homeostasis induces Calpain-Cathepsin axis activation in sporadic Creutzfeldt-Jakob disease.
第一作者:
Franc,Llorens
第一单位:
Department of Neurology, University Medical Center Göttingen, and German Center for Neurodegenerative Diseases (DZNE), Robert Koch Strasse 40, 37075, Göttingen, Germany. franc.llorens@gmail.com.;German Center for Neurodegenerative Diseases (DZNE), Translational Studies and Biomarkers, Site Göttingen, Germany. franc.llorens@gmail.com.;CIBERNED (Network center for biomedical research of neurodegenerative diseases), Institute Carlos III, Ministry of Health, Madrid, Spain. franc.llorens@gmail.com.
作者:
医学主题词
动物(Animals);脑(Brain);钙(Calcium);卡配因(Calpain);组织蛋白酶类(Cathepsins);阳离子, 二价(Cations, Divalent);细胞, 培养的(Cells, Cultured);克-亚综合征(Creutzfeldt-Jakob Syndrome);疾病模型, 动物(Disease Models, Animal);内环境稳定(Homeostasis);人类(Humans);溶酶体(Lysosomes);金仓鼠(Mesocricetus);小鼠, 转基因(Mice, Transgenic);神经元(Neurons);PrPSc蛋白质类(PrPSc Proteins);大鼠, Wistar(Rats, Wistar);重组蛋白质类(Recombinant Proteins);绵羊(Sheep)
DOI
10.1186/s40478-017-0431-y
PMID
28449707
发布时间
2018-11-13
- 浏览21
Acta neuropathologica communications
2017年5卷1期
35页
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