Oxidative Stress Resulting From <i>Helicobacter pylori</i> Infection Contributes to Gastric Carcinogenesis.

导出 LinkOut

翻译打印收藏纠错

作者： Lindsay D,Butcher ^[1] ; Gerco,den Hartog ^[1] ; Peter B,Ernst ^[2] ; Sheila E,Crowe ^[1]

作者单位： Department of Medicine, University of California, San Diego, La Jolla, California. ^[1] Department of Pathology, University of California, San Diego, La Jolla, California. ^[2]

关键词 AP Endonuclease APE1, apurinic/apyrimidinic endonuclease 1 BabA, blood group antigen binding adhesion CagA, cytotoxin-associated gene A DNA Damage Gastric Cancer H pylori IL, interleukin NADPH, nicotinamide adenine dinucleotide phosphate NapA, neutrophil activating factor A Nox, nicotinamide adenine dinucleotide phosphate oxidase O2-, superoxide OH, hydroxyl radical Oxidative Stress RNS, reactive nitrogen species ROS, reactive oxygen species TGF-β, transforming growth factor β VacA, vacuolating cytotoxin A iNOS, inducible nitric oxide synthase

DOI 10.1016/j.jcmgh.2017.02.002

PMID 28462373

发布时间 2022-04-08

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览82

Cellular and molecular gastroenterology and hepatology

2017年3卷3期

316-322页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Oxidative Stress Resulting From <i>Helicobacter pylori</i> Infection Contributes to Gastric Carcinogenesis.

Cellular and molecular gastroenterology and hepatology

相似文献

Cellular and molecular gastroenterology and hepatology

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Oxidative Stress Resulting From <i>Helicobacter pylori</i> Infection Contributes to Gastric Carcinogenesis.

Cellular and molecular gastroenterology and hepatology

相似文献

Cellular and molecular gastroenterology and hepatology

相关期刊

检索历史清除