bFGF Protects Against Oxygen Glucose Deprivation/Reoxygenation-Induced Endothelial Monolayer Permeability via S1PR1-Dependent Mechanisms.

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作者： Li,Lin ^[1] ; Qingzhi,Wang ^[2] ; Kenny,Qian ^[2] ; Zeyuan,Cao ^[3] ; Jian,Xiao ^[2] ; Xiaoying,Wang ^[2] ; Xiaokun,Li ^[1] ; Zhanyang,Yu ^[2]

作者单位： School of Pharmaceutical Sciences, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou, Zhejiang, 325035, China. ^[1] Neuroprotection Research Laboratory, Departments of Radiology and Neurology, Massachusetts General Hospital and Harvard Medical School, 149 13th Street, Room 2401, Charlestown, Boston, MA, 02129, USA. ^[2] Zhengzhou University First Affiliated Hospital, Zhengzhou, Henan, 450000, China. ^[3] School of Pharmaceutical Sciences, Key Laboratory of Biotechnology and Pharmaceutical Engineering, Wenzhou Medical University, Wenzhou, Zhejiang, 325035, China. xiaokunli@163.net. ^[4] Neuroprotection Research Laboratory, Departments of Radiology and Neurology, Massachusetts General Hospital and Harvard Medical School, 149 13th Street, Room 2401, Charlestown, Boston, MA, 02129, USA. Yu.Zhanyang@mgh.harvard.edu. ^[5]

关键词 Basic fibroblast growth factor (bFGF)Endothelial cell monolayer permeability FGF receptor 1 (FGFR1)Junction protein Oxygen-glucose deprivation and reoxygenation (OGD/R)Sphingosine-1-phosphate receptor 1 (S1PR1)